Nicotine is a human teratogen linked to prenatal morbidity, sudden infant death, attention deficit hypersensitivity disorder (ADHD), among other deficits in cognitive function, learning, and memory. Past studies have suggested that these defects result from nicotine-induced depolarization of key ion channels and subsequent disruption of bioelectrical signaling in the embryonic cells.
A new study now demonstrates that such signals can be predicted, mapped, and manipulated to rescue developmental brain damage. The published findings in Nature Communications come from a research team at Tufts University. Read more